Candida, Hashimoto’s, Hypothyroidism, and Hormones

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Candida (yeast) is a type of fungus that naturally lives in our bodies. Numerous different strains can cause infections in the human body, but the most common is Candida albicans, which is usually a harmless part of our gut microbiome. Under normal conditions, this type of candida is an essential member of the normal gut flora and helps maintain balance between bacteria and yeast. But when conditions in the body change—like during periods of stress, illness, hormonal shifts, or after taking antibiotics—Candida can multiply out of control and become overgrown. That’s when it can turn from a friendly resident into an opportunistic pathogen and troublemaker, triggering symptoms that range from digestive upset to fatigue, brain fog, and even immune system imbalances.

Hashimoto’s disease is an autoimmune disease that is the most common cause of hypothyroidism – an underactive thyroid with low thyroid hormone levels – in the U.S. For people with Hashimoto’s, the immune system, hormones, and gut health are closely intertwined. When one aspect is off, the others often follow. While scientists haven’t proven that candida causes Hashimoto’s disease, growing research suggests that Candida overgrowth may worsen thyroid autoimmunity or amplify the thyroid hormone imbalance symptoms—especially during times of significant hormonal change like perimenopause and menopause.

Let’s explore what the latest research says about the impact of Candida on Hashimoto’s thyroiditis, thyroid dysfunction, hormones, and overall well-being—and what you can do to restore balance.

When candida becomes overgrown, infections can occur in various parts of the body, including the:

• Gut
• Mouth
• Nasal cavity
• Vagina
• Rectum
• Skin folds

In rare cases, candida overgrowth may spread into the bloodstream and reach the brain, heart, liver, spleen, bones, muscles, joints, and eyes.

Candidiasis, another term for yeast overgrowth, may cause a variety of symptoms. People with yeast overgrowth in common areas like the mouth and vagina may experience burning and itching in those areas. Often, you can also see evidence of white overgrowth, known as thrush.

Aside from these common symptoms, candida overgrowth may also play a role in:

• Infertility
• Fatigue
• Brain fog
• Weight gain
• Weakness and muscle pain
• Gas, bloating, diarrhea, and constipation
• Muscle and joint pain
• Depression
• Food sensitivities

To see how candida, Hashimoto’s, and hormonal shifts might interconnect, imagine three legs of a stool:

• The immune system (autoimmunity, inflammation)

• The gut environment (microbiome, intestinal permeability)

• The endocrine/hormonal system (thyroid hormones, estrogen, progesterone, cortisol)

When one leg is off balance, the stool can’t stand easily!

Here are key pathways and emerging evidence:

1. Gut barrier, inflammation, and molecular mimicry

One of the most talked-about links is via intestinal permeability (sometimes called “leaky gut”). If candida (or other organisms) overgrow in your gut, they can release toxins or metabolic byproducts. In a susceptible host, those can weaken connections between cells, allowing toxins to enter your circulation. Over time, this can lead to chronic immune activation and low-grade systemic chronic inflammation.

If you’re genetically predisposed, that inflammatory response may cross-react with your own tissues—a phenomenon called molecular mimicry—potentially triggering or worsening autoimmune disease, including Hashimoto’s autoimmune thyroiditis.

While this mechanism is plausible and often discussed in functional-medicine and integrative health circles, it is important to stress that there is no clear proof that candida is a direct cause of Hashimoto’s. Scientists regard candida more as a cofactor or aggravator than a primary cause of thyroid autoimmunity.

2. Immune skewing: Th1/Th2 balance and regulatory T cells

Candida can stimulate various immune pathways. In particular, candida antigens may shift the immune balance toward Th2 responses (allergic/antibody-mediated) or trigger overactivation of specific pathways. Some experts suggest that in Hashimoto’s, a dysregulated immune balance contributes to thyroid autoimmunity. Candida’s presence may aggravate this imbalance.

Again, this remains a hypothesis that hasn’t been proven in clinical trials. But some practitioners believe candida overgrowth may “tilt the scales” if your immune system is already vulnerable due to genetic susceptibility to autoimmunity, viral exposures, or gut health problems.

3. Hormonal influence: Estrogen, progesterone, and thyroid

This is where the “hormones” part really comes in. Several lines of evidence suggest that sex hormones—especially estrogen—can influence candida, your immune function, and microbial balance. In turn, hormonal transitions (perimenopause, menopause) may create an environment more permissive to candida overgrowth.

Here’s a breakdown of relevant findings:

• Estrogen makes candida stronger and harder for your body to fight off: A 2022 paper showed that estrogen can help Candida albicans evade innate immune detection, promoting its survival and growth in tissues. Earlier studies showed that elevated estrogen is associated with higher rates of vaginal candidiasis.

• Menopause triggers increased fungal growth: A recent review in Nature Microbiome discussed how hormonal changes (such as lower estrogen and progesterone levels) can weaken the body’s defenses and shift the balance of microbes, leading to increased fungal growth.

• Estrogen and vaginal pH/colonization: A 2025 study assessed how estradiol (E2) levels correlate with vaginal pH and candida growth, finding that higher E2 levels can correlate with lower, more acidic pH. This weakens the mucosal barriers and microbial “checks and balances” that help control candida.
• Estrogen binding: Some research suggests that estrogen binds to candida, causing it to change from its healthy microbial state to a pathogen. The body mounts a stress response when pathogenic candida is present in your system, which can worsen yeast overgrowth.

Recent research is also looking at the estrobolome, the gut bacteria that metabolize estrogen. When healthy, the estrobolome helps maintain the estrogen balance in your body. But when the estrobolome is imbalanced, your hormonal symptoms can worsen.

4. Thyroid hormones, microbial ecology, and gut transit

Thyroid hormones influence gut motility, digestive secretions, and overall gut health. In hypothyroidism (including in autoimmune hypothyroidism), slower gut transit time is common, which can favor microbial overgrowth, including candida or opportunistic yeasts and bacteria.

Additionally, recent research suggests that shifts in thyroid function (even within the “normal” to subclinical range) are associated with changes in the gut bacteria, which could predispose to candida dominance or dysbiosis.

In short, hypothyroidism may indirectly predispose the gut to slower clearance of pathogens or fungi.

5. Lifecycle events, estrogen dominance, and cumulative stress

Many women with Hashimoto’s struggle with estrogen dominance (relative excess of estrogen compared to progesterone), whether from outside sources (like birth control, hormone replacement) or metabolic drivers (e.g., excess fat tissue, insulin resistance). Excess estrogen is often discussed in integrative literature as a risk factor that enhances candida overgrowth.

Thus, if a woman in perimenopause already carries Hashimoto’s plus estrogen dominance, the hormonal milieu might worsen the conditions for candida to shift from benign to pathogenic.

6. Emerging research and future directions

• A 2025 study on dietary fiber in Hashimoto’s thyroiditis patients explored how fiber intake affects gut bacteria and thyroid autoimmunity, indirectly underscoring the gut–thyroid connection.

• The 2025 Bridging Microbiomes paper discussed how changes in thyroid hormone levels are related to changes in gut bacteria, supporting the idea of a back-and-forth relationship between the gut and thyroid.

While the research doesn’t directly show that candida causes Hashimoto’s, it supports the idea that hormonal shifts, thyroid function, and the makeup of the gut microbiome (including fungi like candida) are strongly related.

Here’s a conceptual scenario illustrating how these components might interplay if you’re a person predisposed to thyroid autoimmunity:

1. Genetic and environmental susceptibility create an underlying risk for autoimmunity

2. Candida overgrowth (due to diet, antibiotic use, hormonal imbalance, etc.) damages the integrity of your gut barrier and provokes chronic immune activation.

3. This immune activation triggers or amplifies your thyroid autoimmunity, worsening Hashimoto’s and leading to hypothyroidism.

4. Hypothyroidism slows gut motility, disrupting your microbial balance and promoting more candida overgrowth.

5. Hormonal transitions (perimenopause, menopause), estrogen fluctuations or imbalances, and declining gut lining mucosal defenses weaken your candida control, allowing overgrowth to worsen.

6. The cycle reinforces itself, creating a “vicious loop” of gut imbalance, immune activation, thyroid stress, hormonal instability, and candida overgrowth.

This model is still primarily theoretical—but many clinicians working at the intersection of gut health, endocrinology, and women’s health use some version of it in practice. The bottom line: Candida overgrowth may not cause Hashimoto’s outright—but in individuals susceptible to autoimmune disorders, it may amplify inflammation, immune dysregulation, and symptoms, especially during hormonal transitions.

Your diet plays a central role in shaping your microbiome. If you have a tendency for candida overgrowth, certain foods and patterns may tip the balance toward increased candida. Let’s take a look at what to avoid, what to moderate, and what supports balance.

Foods and nutrients that promote candida growth

These foods and nutrients are often discussed in antifungal and functional diet protocols for candida. Not all have strong clinical trials behind them, but many are supported by microbial biology, historical use, and case reports. Always personalize with your clinician or nutritionist.

Refined sugars and simple carbohydrates

• White sugar, table sugar, cane sugar, high-fructose corn syrup
• Syrups (maple syrup, agave, molasses) in large amounts
• Sweets, candies, baked goods
• Sugary beverages (sodas, sweetened teas, juice concentrates)

Candida uses glucose, fructose, and other sugars as its energy sources. Excess sugar intake can feed fungal overgrowth.

Refined grains / high-glycemic carbs

• White bread, white rice, pasta, pastries
• Refined cereals and crackers
• Processed flour-based snacks

These convert quickly to sugars in the gut, creating a favorable environment for yeast.

High-starch foods / certain tubers and legumes (in sensitive individuals)

• Potatoes, sweet potatoes, yams (especially in large quantities)
• Corn, peas
• Beans, lentils—especially those that are harder to digest (e.g., if you have underlying IBS or SIBO)

While these foods are nutritious for many, in people with very reactive gut/yeast tendencies, they may worsen candida and fungal growth unless carefully prepared by soaking or sprouting.

Certain fruits / fruit juices / dried fruits

• Especially high-sugar fruits like mango, grapes, cherries, bananas, and pineapple
• Fruit juices or concentrated fruit syrups
• Dried fruits (dates, raisins, figs), which are sugar-dense

In many candida diets, low-glycemic fruits (such as berries and green apples) are allowed in moderation, while high-sugar fruits are limited.

Alcohol and fermented sugary beverages

• Beer, wine, cider
• Liquors with sugar mixers
• Sparkling fruit beverages

Alcohol may alter gut bacteria and “feed” candida growth.

Dairy products with lactose

• Milk (especially when high in lactose)
• Ice cream
• Sweetened yogurt

Lactose is a sugar that some candida species can metabolize. (Note: some protocols allow plain Greek yogurt or kefir as probiotic sources; this depends on your specific tolerance.)

Mold / yeast-prone / fermented foods (in sensitive individuals)

• Bread (especially sourdough, unless it’s well fermented)
• Mushrooms, yeasts, nutritional yeast
• Vinegar, pickles, sauerkraut, kombucha, beer
• Aged cheeses (e.g., blue cheese, brie, Roquefort)

These foods themselves may not cause candida overgrowth in everyone, but some practitioners advise limiting them during an active “candida cleanse” phase.

Sweet condiments / hidden sugars

• Ketchup, BBQ sauce, fruit jams
• Salad dressings with added sugars
• Canned sauces, marinades, and sauces with corn syrup or sugar

Refined oils, saturated fats, and emulsifiers

• Some protocols suggest reducing excess saturated fats or highly processed oils that may impair beneficial microbiota, though the evidence is more speculative. 

Food additives

• Maltodextrin, dextrose, glucose syrup
• Artificial sweeteners, which may support fungal growth or disrupt gut balance
• Emulsifiers (e.g., polysorbate 80) or stabilizers may negatively affect gut barrier integrity

Foods and strategies that help suppress candida

While eliminating or reducing the above, you can focus on foods that support gut resilience and microbial balance:

• Non-starchy, fibrous vegetables: Leafy greens, broccoli, cauliflower, asparagus, Brussels sprouts, zucchini, cucumbers

• Low-glycemic fruits (in moderation): Berries (blueberry, strawberry, raspberry), green apples, and small portions of citrus

• Lean proteins: Eggs, fish, poultry, tofu, tempeh (if tolerated)

• Healthy fats: Olive oil, avocado, coconut (in moderation), nuts/seeds (if tolerated)

• Fermented foods / probiotic foods (carefully introduced): Unsweetened sauerkraut, kimchi, kefir or yogurt (if tolerated), coconut yogurt (sugar-free). Note: Some candida protocols suggest introducing fermented foods only after the “die-off” phase or in stabilization, because they may provoke symptoms in some people.

• Coconut oil, garlic, onion, oregano, ginger, turmeric: Many natural antifungal/antimicrobial foods are used in integrative protocols (e.g., garlic and oregano oil).

• Bone broth/collagen: Supports gut lining health

• Prebiotic fibers: Inulin, resistant starch, partially hydrolyzed guar gum—introduced carefully—can nourish beneficial bacteria that compete with candida.

• Hydration and herbal teas: Water, ginger tea, chamomile, peppermint (if tolerated)

1. Elimination / “reset” phase

• Remove or minimize most of the sugar, refined carbs, alcohol, dairy, and moldy/fermented foods

• Emphasize vegetables, lean proteins, healthy fats, and moderate low-glycemic fruits

• Introduce antifungal foods and herbs (garlic, oregano) under guidance‍

2. Reintroduction/rotation phase

• Gradually reintroduce one food group at a time and monitor symptoms

• Rotate foods so as not to overexpose the gut to one substrate

3. Maintenance / long-term balance

• Keep sugar and refined carbs to a moderate level

• Focus on a diverse, fiber-rich diet

• Include probiotic and prebiotic foods

• Address underlying conditions (thyroid, gut motility, hormonal balance)

Note: Clinical evidence for the “candida diet” is mixed. Some pilot studies suggest that combining diet with antifungal therapy yields better outcomes than antifungals alone. However, large-scale, robust trials are lacking—so these dietary strategies should be part of a comprehensive, individualized plan.

Several common medications can disrupt the delicate balance of microorganisms that live in and on your body—your microbiome—making it easier for candida to multiply. While these drugs are often necessary and beneficial for specific conditions, they can unintentionally create the perfect environment for yeast overgrowth. Here’s how:

Antibiotics

Antibiotics are among the most well-known culprits. They kill off harmful bacteria when you’re fighting an infection, but they also wipe out beneficial bacteria that help keep candida in check. Without those protective microbes, yeast can thrive, especially in the gut, mouth, or genitals. Even short courses of antibiotics can trigger overgrowth in some people, particularly if you already have other risk factors such as immune dysfunction or hormonal imbalance.

Proton pump inhibitors (PPIs)

PPIs—such as omeprazole (Prilosec), esomeprazole (Nexium), and lansoprazole (Prevacid)—are commonly used to treat acid reflux and prevent stomach ulcers. By reducing stomach acid, they can help ease symptoms, but also lower your body’s natural defenses. Stomach acid helps control pathogens and maintain microbial balance, so when acid levels drop, candida can more easily survive and colonize the gut.

Birth control pills and hormone replacement therapy (HRT)

Estrogen-rich medications, including oral contraceptives and certain forms of hormone replacement therapy, can alter the balance of bacteria and yeast—especially in women. Estrogen promotes glycogen production in vaginal tissues, and glycogen provides a food source for candida. That’s why some women notice an increase in yeast infections or digestive candida symptoms after starting or changing hormonal therapies.

Corticosteroids and immunosuppressive drugs

Medications like prednisone or immunosuppressants used for autoimmune diseases and organ transplants can weaken your immune response. A suppressed immune system has a harder time controlling candida, allowing it to multiply unchecked. Inhaled corticosteroids (used for asthma) can also promote candida growth in the mouth if users don’t rinse thoroughly after each use.

Certain diabetes and cancer medications

Drugs that affect your blood sugar levels or immune activity can indirectly fuel candida growth. High blood sugar is a known yeast trigger, so underlying insulin resistance and medications that raise glucose can make yeast thrive. Likewise, chemotherapy drugs or biologics that weaken immune surveillance can create a similar imbalance.

Beyond medications and diet, certain lifestyle habits and environmental factors can make your body more vulnerable to yeast overgrowth. Candida loves conditions that weaken your immune system, alter your hormone balance, or cause chronic inflammation. Here’s a look at the most common non-diet factors that promote overgrowth:

Chronic stress

Your gut is particularly prone to stress, and even minor stressors can throw off your microbiome. When you’re under constant stress, your body releases higher levels of cortisol—the “stress hormone.”  Cortisol not only can increase intestinal permeability but can also suppress immune function and raise blood sugar levels, both of which encourage candida growth. Long-term stress also affects digestion and can alter gut motility, giving yeast more time to multiply in the intestines.

Lack of sleep

Sleep is when your body restores immune balance and repairs tissues. Poor sleep—or too little of it—can weaken the immune system and disrupt the gut-brain axis, a key communication line that helps maintain microbial balance. People who “short sleep” and consistently get less than 7 hours a night are more likely to experience immune dysregulation that makes them prone to infections, including candida overgrowth.

Sedentary lifestyle

Physical inactivity contributes to sluggish digestion, poor circulation, and metabolic slowdown—all of which can indirectly support yeast overgrowth. Regular exercise improves immune surveillance, gut motility, and hormonal balance, helping keep candida under control.

Chronic illness or immune suppression

Autoimmune conditions like Hashimoto’s thyroiditis, diabetes, and other endocrine disorders can impair your immune defenses or alter the microbiome. This makes it easier for candida to take hold. For example, low thyroid function slows metabolism and gut movement, while insulin resistance elevates blood sugar levels—both conditions yeast can exploit.

Environmental toxins and mold exposure

Exposure to household mold, pesticides, or other environmental toxins may alter your immune and liver detoxification systems, reducing your body’s ability to regulate microbial balance. Mold and candida are both fungi, and some experts believe mold exposure can sensitize the body, increasing vulnerability to candida-related symptoms.

Poor hygiene or tight clothing

In areas like the groin, underarms, or under the breasts, candida thrives in warm, moist environments. Wearing tight, synthetic clothing that traps heat and moisture, or failing to dry thoroughly after bathing, can create ideal conditions for local yeast growth.

Recurrent infections and overuse of antifungals

Repeated yeast infections or overuse of antifungal medications can create resistant Candida strains. Over time, this makes the microbiome harder to rebalance naturally, even when other factors improve.

Treating candida overgrowth (especially in the gut) is often more art than science because no single test or protocol is universally accepted. But over the decades, clinicians like those at Paloma Health have developed multi-layered strategies that blend conventional, integrative, and emerging approaches.

Assessment and diagnostic tools

Before starting treatment, many clinicians gather data, while recognizing that testing for candida is imperfect. They assess symptoms such as GI issues, brain fog, fatigue, and recurrent yeast or skin infections. They may use stool tests, urine organic acid tests, and blood antibody panels to detect fungal markers or immune responses. Some also check CBCs and immune markers for signs of dysregulation, though these are non-specific. Most savvy practitioners also ultimately look at symptom patterns and treatment response, beginning therapy empirically and monitoring progress closely.

Eliminate and starve the yeast

As detailed above, dietary measures are foundational. Removing or reducing the foods that feed candida (sugars, refined carbs, alcohol) creates a metabolic challenge for the yeast.

Parallel to diet, lifestyle shifts help:

• Improve sleep quality

• Manage stress (cortisol overactivity can impair immunity and increase gut permeability)

• Regular movement and gentle exercise to enhance gut motility

• Supporting detoxification (liver, bile flow)

• Avoid unnecessary antibiotics, proton pump inhibitors, or medications that disrupt gut flora

• Ensure adequate hydration and fiber intake

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Direct antifungal/antimicrobial agents

Often, the next step is to add agents that reduce candida load. These are used in cycles or pulsed protocols to reduce resistance and minimize side effects.

Conventional antifungals

• Fluconazole (Diflucan) — systemic; widely used for vaginal and systemic candidiasis

• Itraconazole, Voriconazole — more potent, often reserved for severe or refractory cases

• Nystatin — non-absorbed antifungal used for fungal overgrowth in the GI lumen

These are prescription drugs and should be used under physician supervision. They may cause die-off reactions (Herxheimer reactions) when fungal cells release toxins as they die.

Botanical and natural antifungals

Many clinicians use combinations of natural agents, often alternating them to reduce tolerance. Some commonly used ones include:

• Oregano oil, thyme oil (carvacrol, thymol)

• Garlic/allicin / aged garlic extract

• Caprylic acid (from coconut oil)

• Pau d’Arco (Tabebuia impetiginosa)

• Berberine/goldenseal / Oregon grape root

• Berberine hybrids (e.g., berberine and catechins)

• Black walnut hull, wormwood, and garlic combinations

• Grapefruit seed extract

• Pomegranate / ellagic acid

These botanicals may have antifungal, antibacterial, or antiparasitic properties and are often dosed in rotation (e.g., 2 weeks on, 1 week off) or reduced after a “clearance” phase. Because herbal protocols vary widely, working with a trained clinician or naturopath is critical.

Restore and support microbial balance

After reducing the pathogenic fungal load, the next goal is to repopulate beneficial microbes that compete with candida and restore gut resilience.

• Probiotics/beneficial bacteria: Particularly Lactobacillus, Bifidobacterium, Saccharomyces boulardii (a beneficial yeast). Some strains may exert antifungal effects by producing acids or competing for nutrients.

• Prebiotics/fibers: Inulin, fructo-oligosaccharides, partially hydrolyzed guar gum (PHGG), resistant starches (in small, tolerable amounts) help nourish beneficial bacteria.

• Postbiotics/microbial metabolites: Butyrate and short-chain fatty acids (SCFAs) support colonocyte health and barrier integrity.

• Mucosal support nutrients: L-glutamine, aloe, deglycyrrhizinated licorice (DGL), slippery elm, marshmallow root, zinc carnosine, N-acetyl glucosamine — to help heal the gut lining.

• Digestive support: Digestive enzymes, bile acids, and hydrochloric acid (HCl) support proper digestion and reduce the undigested substrate that can ferment in the gut.

Address underlying drivers and reinforce resistance

This is often the most critical and most overlooked component—because if you only suppress the candida but don’t address what allowed it to flourish, relapse is likely.

Some key areas:

• Optimize thyroid therapy: Make sure you’re on the right dose/formulation of thyroid hormone therapy so your metabolism, gut motility, and immune regulation are supported. Regular thyroid blood tests to evaluate thyroid-stimulating hormone (TSH level), Free T4, Free T3, and thyroid peroxidase antibodies (TPO) can help ensure that you stay on an optimal dosage for your thyroid condition. 

• Treat coexisting gut issues: SIBO, IBS, dysbiosis, small intestinal fungal overgrowth (SIFO), H. pylori, bile acid dysregulation—these often coexist and must be assessed.

• Hormonal balance: For women in perimenopause or menopause, balancing estrogen, progesterone, and possibly bioidentical hormones can help restore mucosal integrity, microbial balance, and reduce candida-friendly conditions.

• Detoxification and liver support: As fungal toxins are released, you’ll want to support liver detox, glutathione production, methylation, etc.

• Immune modulation: Address nutrient deficiencies (vitamin D, zinc, selenium, B vitamins), reduce chronic infections, support regulatory T cell function, manage chronic stress, support sleep and circadian rhythm, and reduce environmental inflammatory exposures.

• Lifestyle shifts: Exercise, stress reduction (meditation, breathwork), sleep hygiene, toxin avoidance (e.g., mold, chemicals) all matter.

Maintenance and monitoring

• After the “clearing” and reinoculation phases, most clinicians move to a maintenance regimen, which may include low-dose antifungals (botanical or conventional), intermittent probiotic pulses, dietary moderation, and periodic check-ins.

• Monitor symptom trends, inflammatory markers, stool/fungal panels (if repeated), and thyroid/immunity labs.

• Be alert for die-off reactions (transient fatigue, headache, GI upset). Supportive measures such as hydration, binders like activated charcoal or bentonite clay, and gentle liver support may help.

• Adjust and evolve the plan—what works for 8 weeks may need recalibration later.

Safety, precautions, and caveats

• Potent antifungals (conventional or botanical) can stress the liver and cause side effects—always work with a healthcare provider.

• Die-off reactions may be intense in sensitive individuals—titrate dosages carefully.

• Some “candida overgrowth” diagnoses are over-applied; not every GI or fatigue symptom is due to candida.

• In immunocompromised individuals, systemic candidiasis is a medical emergency and requires specialist care.

• There is a risk of opportunistic resistance if antifungals (especially conventional ones) are overused.

• Always monitor nutritional status: aggressive restriction of food groups or prolonged “elimination diets” may lead to deficiencies (e.g., in B vitamins, fiber, trace minerals).

At Paloma Health, we know how overwhelming it can be to piece together symptoms that don’t seem to fit neatly into one box — fatigue, bloating, brain fog, flare-ups, hormonal shifts, and thyroid changes. If you suspect candida overgrowth may be playing a role in your Hashimoto’s or hormonal symptoms, you’re not alone. Many people spend years searching for answers before discovering how interconnected the gut, thyroid dysfunction, and hormones really are.

Paloma’s care team takes a whole-body approach to thyroid and hormonal health because true wellness rarely comes from treating one piece in isolation. Whether you’re navigating persistent digestive issues, trying to understand unexplained thyroid swings, or looking for support in balancing hormones during perimenopause or menopause, we’re here to help you find clarity — and a plan that actually works for your life.

If you’re ready for more personalized guidance, comprehensive testing, or expert support from clinicians who truly understand hypothyroidism, Hashimoto’s, and hormonal health, consider partnering with Paloma on your journey.

• Candida is a normally harmless yeast that can overgrow during stress, illness, antibiotics, or hormonal changes.
• Candida overgrowth may worsen symptoms in people with Hashimoto’s, but it is not proven to cause the disease directly.
• Hormonal shifts, especially in estrogen during perimenopause and menopause, can encourage candida growth.
• Hashimoto’s disease and hypothyroidism can slow gut motility, indirectly promoting yeast overgrowth and dysbiosis.
• Diet, lifestyle, and medications (like antibiotics, PPIs, or estrogen therapy) can influence candida growth.
• Treatment combines diet, antifungal agents, probiotics, and addressing underlying hormonal and thyroid imbalances.